By Elizabeth Neal
Epilepsy is a standard continual neurological sickness characterized by way of recurrent unprovoked seizures, and impacts 50 million humans around the world. nearly 30% of those do not need seizure regulate regardless of the easiest to be had medicines, for that reason renewed and lengthening curiosity within the use of nutritional remedies to regard epilepsy. those remedies comprise the conventional ketogenic diets but additionally the more recent changed Atkins vitamin and coffee glycaemic index treatment.
The ketogenic nutrition is a high-fat, adequate-protein, low-carbohydrate vitamin that during drugs is used essentially to regard refractory epilepsy in teenagers, and calls for the devoted help of a multi-disciplinary workforce together with medical professionals and dietitians. Dietary remedy of Epilepsy: sensible Implementation of Ketogenic Therapy offers finished and useful education on all points of imposing and handling nutritional remedies for epilepsy, with key chapters on review, calculations, implementation, tracking and complications.
Edited via a hugely revered specialist within the box supported by way of a robust group of members, it is a must-have source for paediatric dietitians and different contributors of the treatment workforce.
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Extra info for Dietary treatment of epilepsy: practical implementation of ketogenic therapy
2009) Optimal clinical management of children receiving the ketogenic diet: recommendations of the international ketogenic diet study group. Epilepsia 50, 304–317. , Turner, Z. and Gladstein, J. (2010a) Use of the modified Atkins diet for adolescents with chronic daily headache. Cephalalgia 30, 1014–1016. C. D. (2010b) Will seizure control improve by switching from the Modified Atkins Diet to the traditional ketogenic diet? Epilepsia 51, 2496–2499. Lefevre, F. and Aronson, N. (2000) Ketogenic diet for the treatment of refractory epilepsy in children: a systematic review of efficacy.
Within the chylomicron, triglycerides are hydrolysed to free fatty acids and glycerol, which can be used by cells or stored in adipose tissue. Some free fatty acids bind to albumin. Free fatty acids bound to albumin can be taken into hepatocytes via the portal circulation. Fatty acids form acyl-CoA derivatives (via long-chain acyl-CoA synthetase). Very long chain fatty acids undergo initial metabolism via β-oxidation in peroxisomes. Metabolism of short-, medium- and long-chain fatty acids proceeds primarily through mitochondrial β-oxidation.
P. M. (2006) The importance of cognition in parental expectations prior to starting the ketogenic diet. Epilepsy Behav 8, 406–410. , Rousselle, C. and David, M. (2003) Ketogenic regime as anti-epileptic treatment: its use in 29 epileptic children [French]. Arch Pediatr 10, 300–306. C. T. (1998) The efficacy of the ketogenic diet 1998: a prospective evaluation of intervention in 150 children. Pediatrics 102, 1358–1363. , Ye, X. N. (2009) A blinded, crossover study of the ketogenic diet. Epilepsia 50, 322–325.
Dietary treatment of epilepsy: practical implementation of ketogenic therapy by Elizabeth Neal